NURS 6501 GASTROINTESTINAL AND HEPATOBILIARY DISORDERS Scenario 2: Gastroesophageal Reflux Disease (GERD) A 44-year-old morbidly obese female comes to the clinic complaining of  “burning in my chest and a funny taste in my mouth”.

The symptoms have been present for years but
patient states she had been treating the symptoms with antacid tablets which helped
until the last 4 or 5 weeks. She never saw a healthcare provider for that. She says the
symptoms get worse at night when she is lying down and has had to sleep with 2
pillows. She says she has started coughing at night which has been interfering with her
sleep. She denies palpitations, shortness of breath, or nausea. 
PMH-HTN, venous stasis ulcers, irritable bowel syndrome, osteoarthritis of knees,
morbid obesity (BMI 48 kg/m 2 ) 
FH:non contributary   
Medications: Lisinopril 10 mg po qd, Bentyl 10 mg po, ibuprofen 800 mg po q 6 hr prn 
SH: 20 PPY of smoking, ETOH rarely, denies vaping    
Diagnoses: Gastroesophageal reflux disease (GERD). 
 
Question:
1.     If the client asks what causes GERD how would you explain this as a provider? 
Your Answer:
The patient in the case study has GERD. I would inform her that several factors cause
GERD. One of the aspects that I will educate her is that GERD is a condition that develops
following the ulceration of the mucosal lining that protects the esophagus. One of the
causes of the disorder is Zollinger-Ellison syndrome, which increases the release of gastric

acid. Zollinger-Ellison syndrome is characterized by the presence of multiple duodenal or
pancreatic tumors that increase gastric acid secretion (Maret-Ouda et al., 2020).
            The other cause of GERD that the patient should be aware is the prolonged use of
NSAIDs. NSAIDs inhibit the synthesis of protective prostaglandins. They also lower the
production of bicarbonates and mucus while increasing the secretion of hydrochloric acid.
The other factor is smoking. Smoking suppresses the production of prostaglandins, mucus
for protection, and weakens the esophageal sphincter. Increased use of irritants such as
coffee and alcohol also play a crucial role (Katz et al., 2022). The irritation acts as a source
of stress that degrade the protective mucosa and increase the production of destructive
gastric acid.
The other cause is any form of stress. Stressors such as hospitalization and life
experiences also act as a source of GERD. Any stressors increase the production of gastric
acid. The risk of GERD increases if the patient already has other risk factors for GERD and
or peptic ulcer disease. The additional risk factors that should be addressed to prevent
GERD include obesity, hiatal hernia, esophageal contractions, prolonged or reduced
stomach emptying, and abnormalities of esophageal sphincter (Maret-Ouda et al., 2020).

References

Katz, P. O., Dunbar, K. B., Schnoll-Sussman, F. H., Greer, K. B., Yadlapati, R., &Spechler, S. J.
(2022). ACG Clinical Guideline for the Diagnosis and Management of Gastroesophageal
Reflux Disease. The American Journal of Gastroenterology, 117(1), 27–56.
https://doi.org/10.14309/ajg.0000000000001538
Maret-Ouda, J., Markar, S. R., & Lagergren, J. (2020). Gastroesophageal Reflux Disease: A
Review. JAMA, 324(24), 2536–2547. https://doi.org/10.1001/jama.2020.21360Links to
an external site.